Surveys that have been carried out by the healthcare agencies shows an increased cases of this neurological condition in relatives of probands with a notable 50% unaffected relatives who are asymptomatic and when diagnosed shows no sign of JME and therefore considered to be JME negative but PSW-positive. Scientific investigations and findings have established that JME estimations show a likelihood of genetic cause that is found in clustered lineage of families. It is imperative that genetic analysis is undertaken to allow for easy identification and JME disease. Identification of the primary epileptic abnormality is enabled by EEG endophenotype (PSW) and other factors may contribute to the causal relationship. Primary epileptic abnormality identification is instrumental in uncovering how the electric discharges are generated and the explanation behind the cases of clinical seizure in some patients and resistance in others. The overall aim of this study is to give a comprehensive explanation of the occurrence of EEG endophenotype PSW with a sample of a statistically well-defined population. The population for this study constitutes the asymptomatic relatives of JME with a positive EEG-PSW of age ranging 10-40 years. This age group constitutes the old and the young so that there is an exhaustive generalization of this condition’s implications. This action will help in finding out answers to whether the first-degree relative who is EEG-PSW positive and a JME-patient can develop epilepsy over time.