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The vibrant of prostatic obstruction accounts for the variable nature of BPH symptoms experienced by patients (Kadmon, 2011). The prostatic stroma comprised of collagen and smooth muscle is rich with adrenergic nerve supply. Autonomic stimulation, therefore, sets the tone to prostatic urethra. Alpha-blocker therapy whenever administered helps to decrease the outlet resistance (Beckman &amp. Beliveau, 2005).
The irritating voiding complaints of BPH often result from the response of the bladder to increased outlet resistance. Obstruction in the bladder outlet causes detrusor muscle hypertrophy, hyperplasia, and collagen deposition. The latter, is seemingly the most likely responsible cause of decrease in bladder compliance (Rind, 2006). Remarkably, though, detrusor instability is a major factor as well. A detailed inspection reveals that thickened muscle bundles are perceived as trabeculationin in cystoscopic examinations. If unchecked, mucosal herniation ensues between the detusor muscles leading to the formation of Diverticular. The result is in fact the false Diverticular comprised solely of mucosa and serosa (Bushma, 2011). Common clear obstructive symptoms of BPH include hesitancy, reduced force in quality of the stream, sensation of an incomplete emptying of the bladder, double voiding, post void dribbling, and strain when urinating. Irritative symptoms include nocturia, urgency, and frequency (Rind, 2006).
M.P. who is in mid 40s has his father already diagnosed with BPH, implying that he may develop the same. The condition is hereditary. He is overweight as much as he considers himself healthy probably because he has no evident allergies. His wife, who already sensed that M.P. could have been unhealthy, suggested that he seeks medical intervention. His urinary symptomatology includes difficulties in starting his stream of urine, irritation when urinating, nocturia, lower back, and pelvic discomfort. These symptoms