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The individual cannot breakdown such proteins leading to their accumulation and final death of the host as the proteins demand for more space.
Viroids contain RNA as the nucleic acid and have no protein coat. Their infection materializes in the company of viruses. A case example is the viroid, Hepatitis D usually evident in the caspid of the virus, Hepatitis B.
Bacteria are ubiquitous and unlike viruses, they lack the membrane enclosing the DNA contained in their nucleus. This makes them reproduce quickly via binary fission, independent of other cells. Bacteria also adapt to new environments and replicate even faster to overwhelm the hosts immunity. Consequently, a disease is evident.
Viruses in the lytic cycle attach their phage on the host cells using tail fibers. Their penetration occurs once the lysozyme cell wall opens up allowing the tail sheath to contract and enforce DNA and tail core inwards (Harvey, 2007). In this cycle, production of phage proteins and DNA transpires, followed by mellowing and assembly of phage particles prior to their release once the cell wall of lysozyme breaks. Lytic cycles lead to lysis and eventual death of host cell. Chemical bonding and signaling to produce enzymes regulate the process. Latent cycle also known as lysogeny and temperance marks a period of no virulent activity. In this case, prophage DNA may be integrated in the hosts nucleic acid inducing specialized transduction and phage conversion in the future (Tortora, Funke amp. Case, 2010). This results to a provirus that allows infected cells to function normally for some time. The cycle involves the production of repressor proteins, which in spontaneous occasions may fail, making the virus to enter the lytic cycle.
Viruses act as co-carcinogens, in that they propagate cancer progression. In some cases, they cause cancer, though pathology results after a long time. It is said that 10% of